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On Parity Violation in Hadron Physics

Matti Pitkänen

Abstract


Strong interactions involve small CP violation revealing in the physics of neutral kaon and B meson. An interesting question is whether CP violation and also P violation could be seen also in hadronic reactions. QCD allows strong CP violation due to instantons. No strong CP breaking is observed, and Peccei-Quinn mechanism involving axion as a new but not yet detected particle is hoped to save the situation. The de-confinement phase transition is believed to occur in heavy nucleus collisions and be accompanied by a phase transition in which chiral symmetry is restored. It has been conjectured that this phase transition involves large P violation assignable to so called chiral magnetic effect (CME) involving separation of charge along the axis of magnetic field generated in collision, chiral separation effect (CSE), and chiral magnetic wave (CMW). There is some evidence for CME and CSE in heavy nucleus collisions at RHIC and LHC. There is however also evidence for CME in proton-nucleus collisions, where it should not occur. In TGD instantons and strong CP violation are absent at fundamental level. The twistor lift of TGD however predicts weak CP, T, and P violations in all scales and it is tempting to model matter-anti-matter asymmetry, the generation of preferred arrow of time, and parity breaking suggested by CBM anomalies in terms of these violations. The reason for the violation is the analog of self-dual covariantly constant Kahler form J(CD) for causal diamonds CDM4 defining parallel constant electric and magnetic fields. Lorentz invariance is not lost since one has moduli space containing Lorentz boosts of CD and J(CD). J(CD) induced to the space-time surface gives rise to a new U(1) gauge field coupling to fermion number. Correct order of magnitude for the violation for K and B mesons is predicted under natural assumptions. In this article the possible TGD counterparts of CME, CSE, and CMW are considered: the motivation is the presence of parallel E and B essential for CME.

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